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Cancer pathwayKidney / Upper Tract

Renal Mass Approach

A renal mass is sorted first into cystic vs solid and then benign vs malignant — most are incidental, and management runs from surveillance to nephron-sparing surgery.

Sort
cystic vs solid
+
Risk
benign vs malignant
+
Spare
the nephron
Orientation

The big picture

Every renal mass faces two questions: is it cystic or solid, and is it benign or malignant? Cystic lesions are graded by the Bosniak system (higher grade = higher malignancy risk); solid enhancing masses are renal cell carcinoma until proven otherwise.

Golden rule

Solid enhancing mass = RCC until proven otherwise; characterise (Bosniak for cystic), risk-stratify, and spare the nephron whenever oncologically safe.

Pathophysiology

Mechanism pathway

Tap any step to see why it happens.

Work-up

Diagnostic algorithm

Each step answers one question. Tap to expand.

Classification

Classification & subtypes

Simple cyst (Bosniak I–II)

Thin-walled, non-enhancing fluid lesions.

Why it matters:
Benign; no follow-up needed for I–II.
Management:
Reassure; IIF gets surveillance.
Memory hook:
Thin, no enhancement = benign cyst.
Board trap:
Over-investigating a simple cyst.

Complex cyst (Bosniak III–IV)

Thick walls, septa or nodular enhancement.

Why it matters:
Rising malignancy risk with grade.
Management:
Surgery or surveillance by grade/fitness.
Memory hook:
Enhancing septa/nodule = treat.
Board trap:
Calling an enhancing complex cyst benign.

Solid enhancing mass

Enhances after contrast.

Why it matters:
RCC until proven otherwise.
Management:
Surveillance/biopsy/ablation/partial/radical by size and fitness.
Memory hook:
Solid + enhances = RCC until proven otherwise.
Board trap:
Assuming a solid mass is benign without work-up.

Fat-containing mass

Macroscopic fat on CT (no calcification).

Why it matters:
Suggests benign angiomyolipoma.
Management:
Surveillance; treat large/bleeding AMLs.
Memory hook:
Macroscopic fat = think AML.
Board trap:
Fat-poor AML can mimic RCC.
Management

Treatment ladder

Match intensity to malignancy risk and patient fitness while preserving renal function.

1
Characterise + risk-stratify
2
Simple cyst → reassure
3
Small/indolent or frail → surveillance ± biopsy
4
Small mass → partial nephrectomy or ablation
5
Large/central → radical nephrectomy
Procedures

Procedure chooser

Surgical / procedural options
  • Partial nephrectomy (preferred) for localised tumours
  • Radical nephrectomy for large/central tumours not amenable to sparing
Surveillance

Follow-up

What to monitor
  • Mass size/growth on surveillance imaging
  • Renal function
  • Recurrence after treatment
Timing
  • Interval imaging for surveillance; protocol-based post-treatment
Success looks like
  • Stable small mass on surveillance, or treated with preserved function
Failure looks like
  • Growth/progression on surveillance, or recurrence
When to image
  • Per surveillance protocol; new symptoms
Long-term issues
  • CKD risk after nephron loss; recurrence; metachronous lesions
Recall

Memory hooks

Cystic vs solid, then benign vs malignant.

Enhancement = the key sign.

Solid enhancing = RCC until proven otherwise.

Macroscopic fat = AML.

Spare the nephron.

Exam

Board traps

Bosniak III/IV cyst with enhancing components treated as benign.

Small mass in a solitary kidney removed radically.

Fat-poor AML mistaken for, or distinguished from, RCC.

Apply

Clinical cases

Case 1

A healthy 58-year-old has an incidental 3 cm solid, enhancing lower-pole renal mass with a normal contralateral kidney.

What is the working diagnosis and the preferred treatment?

Test yourself

Quiz

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