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Uro-Oncology · Prostate CancerUro-oncology / Prostate

Prostate Cancer: Treatment by Risk

Once you've classified the risk, treatment follows a logic: low-risk disease is often best NOT treated immediately, while intermediate and high-risk disease are treated with surgery or radiation ± hormones.

Orientation

The big picture

Once you've classified the risk (previous lesson), treatment follows a logic: low-risk disease is often best NOT treated immediately (active surveillance spares men the harms of overtreatment), while intermediate and high-risk disease are treated with surgery or radiation ± hormones. The whole field turned on realising that many prostate cancers are indolent — so the art is matching the intensity of treatment to the aggressiveness of the cancer and the life expectancy of the man.

Golden rule

The framework: the options (active surveillance, watchful waiting, radical prostatectomy, radiotherapy ± ADT, ADT) → a chooser by risk group + life expectancy → advanced/metastatic disease.

Pathophysiology

Mechanism pathway

Tap any step to see why it happens.

Illustration

Interactive — systemic therapy by class (EAU)

Work-up

Diagnostic algorithm

Each step answers one question. Tap to expand.

Procedures

Procedure chooser

Risk group / life expectancy

Active surveillance

Fits
Preferred: low (esp. GG1); select favourable-intermediate

Monitoring (PSA, exam, repeat MRI/biopsy) with curative treatment held in reserve if the cancer progresses. Preferred for most low-risk disease to avoid overtreating indolent cancer; may still be appropriate for selected favourable-intermediate men.

Radical prostatectomy (± PLND)

Fits
Option from low; definitive for unfavourable-intermediate / high (PLND in high)

Remove the whole prostate + seminal vesicles (open/laparoscopic/robotic); nerve-sparing where oncologically safe. An option for low/favourable-intermediate men who prefer or are unsuitable for surveillance; definitive for unfavourable-intermediate and high risk (often with pelvic lymph-node dissection in high-risk disease).

Radiotherapy ± ADT

Fits
Option from low; + ADT in higher risk (short-course int., long-course high)

External beam (EBRT) or brachytherapy. An option in low/favourable-intermediate disease (± short-course ADT); given with ADT in unfavourable-intermediate and high risk — long-course ADT in high risk. Neoadjuvant/concurrent/adjuvant ADT improves higher-risk RT outcomes.

Watchful waiting

Limited life expectancy / low threat (palliative)

Less intensive observation with palliative intent (no curative treatment planned), for men with limited life expectancy where the cancer is unlikely to threaten them.

Focal therapy — NanoKnife / IRE (investigational)

Fits
Selected localized, lower-volume / intermediate-risk, MRI-visible disease

Irreversible electroporation (NanoKnife) — a NON-thermal focal ablation (short direct-current pulses → permanent membrane nanopores → cell death) that may better spare adjacent neurovascular bundles and the urethra. One of several focal modalities (alongside HIFU and cryotherapy); investigational — needs larger cohorts / longer follow-up, and is not a replacement for radical prostatectomy or radiotherapy in standard practice.

Life expectancy matters: don't aggressively treat a low-threat cancer in a man unlikely to live long enough to benefit. ADT is given with radiotherapy in higher-risk disease (short-course for intermediate, long-course for high). The verbatim guideline KEY is kept in the mechanism card above.

Exam

Board traps

Low-risk → active surveillance is preferred (avoid overtreating indolent cancer).

High-risk → aggressive multimodal therapy (RP + PLND, or RT + long-course ADT).

Radiotherapy for intermediate/high risk is given ± ADT (improves higher-risk outcomes).

Life expectancy drives intensity — watchful waiting for low-threat cancer in limited life expectancy.

mHSPC: ADT alone is no longer enough — intensify with ARPI ± docetaxel.

Rising PSA after definitive therapy = biochemical recurrence → restage (PSMA PET) + salvage.

Focal therapy: HIFU and cryotherapy are THERMAL; NanoKnife / irreversible electroporation (IRE) is NON-thermal (short DC pulses → permanent membrane nanopores → cell death) — its theoretical advantage is sparing connective tissue and adjacent structures (nerves, vessels, urethra) → better functional outcomes.

IRE/NanoKnife is focal therapy for selected localized disease and remains investigational (needs larger cohorts / longer follow-up) — not standard of care, not a replacement for RP or radiotherapy.

Test yourself

Quiz

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